EMG results
I had a EMG done 11 months ago. It came back abnormal I guess with three different areas.
Now a senior neurologist at a major hospital diagnosed me with ALS. I wasn’t even given a new EMG to show any changes. Just diagnosis and Calvary hospital.
I still don’t believe it and feel it is wrong.
I have hyperreflexia spasticity the babinski sign.
I just want to feel like my left arm isn’t getting weaker. I rode on my bike the other day and carried a big mirror home from a store. Let’s just say my right hand vastly outperformed my left hand. I wonder if this EMG was a one off and a new one could disprove the diagnosis.
Here is the EMG results for anyone interested
R median DII and ulnar DV, normal
R radial sensory, normal
R median and ulnar motor, normal
R sural and superficial peroneal, normal
R peroneal EDB, normal
R tibial motor and F waves, normal
Needle EMG of the right lower extremity in sampled muscles innervated by the L4-S1 nerve roots showed decreased recruitment of large amplitude and long duration MUAPs in the gastrocnemius.
Brief fasciculations were seen in the gastrocnemius.
Needle EMG of the right and left thoracic paraspinal was normal with no abnormal spontaneous activity.
Needle EMG of the right upper extremity in sampled muscles innervated by the C5-C8 nerve roots showed decreased recruitment of large amplitude and long duration MUAPs in the FDI. There was also normal recruitment of large amplitude and long duration MUAPs in the EIP.
Brief fasciculations were seen in the FDI.
Needle EMG of the right trapezius (CN XI) showed brief fasciculations on spontaneous activity with normal recruitment of normal MUAPs on activation.
Needle EMG of the right tongue (CN XII) was normal. No abnormal spontaneous activity was seen.
Conclusion: This is an abnormal study. There is electrodiagnostic evidence of the following:
Active and chronic denervation in lumbar and cervical myotomes as well as active denervation in CN XI innervated muscles consistent with a primary axonal loss process involving motor roots versus anterior horn cells. With involvement of 3 body segments along with upper motor neuron signs on examination, these findings raise concern for amyotrophic lateral sclerosis (ALS). Per Awaji protocol, he meets criteria for clinically possible amyotrophic lateral sclerosis. referred for MND. Fasciculations, cramping, upgoing toes.Twit CT ching in the RUE (Dec 2024) and RLE (March 2025)