u/sporknitebattlepass

I wasn’t sure where else to ask about this. I’ve been wondering a few things about bicalutamide.

So to my understanding, bicalutamide does not work by lowering overall T, but instead by blocking androgen receptors.

I would assume this leaves testosterone to be bound to SHBG, which also increases free estradiol percentage.

Is that assumption correct?

Would this result in more bioavailable E2 than monotherapy?

Dudes Rock.