u/Maximum_Taco_Flight

What happened with obstruction? Or no obstruction? Anyone else have similar results?

Feb 26, 2026
Hospital echo
LVOT gradient 38 mmHg at rest. This was interpreted as obstruction and helped lead to the initial obstructive HCM/HOCM picture.

April 2026
Echo
No significant obstruction documented.

Apr 24–30, 2026
Cardiac MRI
Showed chordal SAM + flow acceleration through the LVOT, so there were signs of disturbed/accelerated outflow. But it also showed something important: mid-to-apical cavity obliteration — the thick heart muscle was squeezing the chamber nearly closed farther down inside the LV.

June 24, 2026
Exercise echo/CPET
About 12 mmHg at rest → 18 mmHg at peak exercise. The official interpretation was essentially no significant LVOT obstruction, including with exercise.

Aug 2026 — Mayo
Mayo evaluation
Mayo did not find the classic LVOT obstruction pattern. Instead, they described mid-ventricular flow acceleration — meaning the narrowing/acceleration appears to originate inside the middle portion of the left ventricle, rather than at the LV outflow tract.

Also 27mm, <15% Fibrosis, NSVT

Most recent MRI says “Severe HCM with a mid-ventricular phenotype/flow acceleration, rather than classic LVOT obstruction.”

So is that “obstructive HCM” or no? Waiting on my next apt., but concerned Camzyos is out of the picture for me.

Headed to Mayo Clinic soon. Sounds like they are going to run a lot of tests in the first few days and then hopefully I’ll have some answers. Has anyone been to Mayo?

I never imagined I would be asking for help like this, but I have reached a point where I need support. Earlier this year, I was unexpectedly diagnosed with hypertrophic cardiomyopathy (HCM), a serious heart condition where the heart muscle becomes abnormally thick and can affect how the heart functions. After a hospitalization, extensive testing, and appointments with specialists, I have been referred to Mayo Clinic

I’m pretty let down by fundraising to go to Mayo Clinic. I actually can’t even believe the amount of my own family who won’t even share the dang post on social media. Like seriously- my own blood. I’m blown away. Heart broken in more way than one.

My cardiologist is sending me to an HCM Specialist…the appointment is 33 days away. I’m freaking out. Just got my MRI back….

Overall, findings are consistent with significant left ventricular hypertrophy, with asymmetric severe septal hypertrophy, maximal wall thickness of 2.7 cm. Based on T1 mapping and GE images there appears to be diffuse fibrosis, LV LGE quantification is challenging due to lack of normal reference segment. There is mid and apical cavity obliteration, chordal SAM with flow acceleration in the LVOT.
Overall, this degree of hypertrophy, fibrosis is more concerning for hypertrophic obstructive cardiomyopathy rather than hypertensive heart disease.
Recommend referral to HCM clinic for further evaluation. Diffuse mid myocardial late gadolinium enhancement, RV insertion site late gadolinium enhancement. LV LGE quantification is challenging due to lack of adequate normal) segment.
T1 mapping values are elevated, consistent with interstitial fibrosis.
LVEF: 60 %
Findings:
LVEF: 60% RVEF: 72 %
Normal left ventricular volume index, systolic function, and wall motion. LVIDd: 4.7 cm. Asymmetric severe basal septal hypertrophy, maximal wall thickness of 2.7 cm in the basal septum. Adjacent basal lateral segment measures 1.4 cm. There is mid to apical LV cavity obliteration, chordal SAM with flow acceleration in the LVOT. Global peak wall thickness 27mm.