r/EKGs

I'm new to interpreting EKGs and I wanted to know if someone can help me. I'm guessing some type of hypokalemia or something like that. Maybe the EKG is not properly done. My teacher told me to solve it

It looks like mobitz 2, but I don't see an atrial depolarization prior to the pause. Is that a necessary component?

86 year old male called for difficulty breathing. Respiratory rate was >40, wet lung sounds audible without auscultation, hypertensive. No hx of heart failure, no peripheral edema noted. Initially rate was variable between 150-170. Pads were applied, CPAP and nitro administered. Rate climbed to 200, pressure remained high. The patient became obtunded and they were synchronized cardioverted.

The rhythm was >.12ms and had a short PRI. My rhythm DDx were atrial flutter with LBBB, vtach, and some sort of WPW. Figured the patient was experiencing cardiogenic pulmonary edema. Im just a newer medic looking for a little help.

Intermittently has this rhythm and then normal sinus with rates in the 80s with NSR. Is this intermittent third degree or second degree type two?

76 YOF w/ no cardiac history. Advised she felt like her heart was racing. During assessment she became extremely distressed, complained of SOB and said she felt like her legs were numb. Second pic was after cardioversion at 50j

Patient has no complaints at the time of the inspection. He notes shortness of breath during physical exertion, denies anginal pain, including in the previous period. According to the ECG from 05/18/2025, no significant dynamics were detected on the ECG series. Troponin T: 23.2 pg/ml → 20.95 pg/ml According to the EchoCG data: no areas of violation of local contractility were detected, global myocardial contractility was preserved, LVEF 58% Is that Wellens? In my opinion, a typical picture of Wellens syndrome is unlikely due to the absence of anginal syndrome and the characteristic dynamics of the ECG. However, the presence of a critical lesion of permanent residence is not completely excluded.

had bronchitis

sick with viral illness (rhino)

Felt dizziness and heart palpitations

Got to the hospital and was in normal sinus rhythm

cardio / EP started flecainide

Hello.

English is not my first language, I don't understand the anology of using the cove to describe brugada type 1 pattern.

I searched for the term "cove" it turns out it mean an area of sea that is partly surrounded by land, but I still did not get it😅

Could anyone explain it to me please?

40yo female w/ no PMHx, no recent complaints, suddenly collapsed, cardiac arrest.

This 12 lead was taken ~6min post-ROSC.

79 yom, recent episodes of being tired, lied down after lunch, woke up hour late with nausea, sweaty, HR between 47-51, ecg showed lbbb, while on way in sudden tachycardia, ecg as shown, lasted about 1 minute.

Everyone seems to be unsure on what rhythm we are seeing here

Hallo zusammen,
ich würde mich über eine fachliche Einschätzung und Interpretation dieses EKGs freuen.
Der Patient war während der Episode wach und ansprechbar.

64 year old female calls EMS for acute onset of shortness of breath that began three hours prior. Upon arrival she was alert oriented times four, tachypneic, and anxious.

Patient reported she’s had three episodes like this prior. Reported she’s received dilt and adenosine in the past. However, she is unable to recall if she had history of SVT or A-fib. But was scheduled to for an ablation soon. No other pertinent medical history.

Vitals 95/50, R:24 SPO2: 98%, HR: 195-200.
RX: carvedilol.

Vasovagal maneuvers unsuccessful. Patient was given 2 mg of Versed for sedation and fluid bonus of lactated ringers. Synchronized cardioverted at 100j successfully.

Other than the discomfort from cardio-version, patient reported overall relief of everything else. . Post vital signs: HR: 128, Bp: 136/75, SPO2: 98% R:22. Patient transported without incident to local hospital.

Woman with PMx of severe kidney failure, heart failure, ablation due to AF and been electrocardioverted x3 for afib/aflutter before. Now initially felt like she had atrial fibrillation for three hours before acute onset of central chest tightness.

I honestly have no idea what kind of rhythm this is. Have clear P-waves that seem dissociated from the QRS-complexes, but there are multiple identical PR-intervals. Is this some sort of conduction block?

A few months ago, my colleagues sent me an ECG of a patient who was in the internal medicine wards and was diagnosed with meningitis complicated by diabetic ketoacidosis (DKA). They asked me what I thought about the ECG.

I immediately asked whether they had the lab results, particularly the electrolyte levels, but they said they hadn’t seen them. The next day, I went to the internal medicine wards, but unfortunately, the patient had been discharged. I tried to ask the physicians, particularly about the potassium levels, and one of the residents told me that the patient had hypokalemia. I asked him if he was sure because the ECG showed peaked T waves. He responded that those T waves were normal, and he said the only abnormality on the ECG was sinus tachycardia.

I have seen several cases of hypokalemia causing peaked T waves; however, in all those cases, the QT interval was significantly prolonged, which was not the case in this ECG.

What do you think? Could this be hyperkalemia or hypokalemia?

Unfortunately, I have no more information about the patient.

59 female. What do you think is happening here?

Patient had 170 troponin and 9/10 chest pain (given morphine then switched to dilaudid). This is second EKG that prompted ER physician to call the on call IC. First ekg had some st elevation and abnormalities but not as bad as this one. On call IC said ‘does not meet any criteria for STEMI’ … 🤨

CKD patient in the ICU, sedated. No cardiac symptoms. Wellens Syndrome? Or just LV strain?

I try searching for pseudo r waves in v1 for avnrt vs Frank spikes in the t wave or just outside qrs complex in v1 for avnrt in narrow complex regular rhythm tachycardias. But in this ecg for instance, i couldn't find either in the lead v1. So is it directly assumed that the p wave was too close to qrs and just fell into it?