Everyone talks about what they added. Less gets said about what came off the list.

Sometimes it is the evidence. You go looking for the human data and find three mouse studies and a forum post. Sometimes it is the opposite, the data is fine but the practical side does not work. Sometimes a better option in the same category showed up and the first one just became redundant.

I am interested in the reasoning more than the compound.

What was it for you, and what actually tipped it? Was it something you read, something you noticed, or just the realization that it was not doing what the writeups said it would?

Hello I’m looking at stacking KPV GHK BPC-157 TB-500 (KLOW) and CJC no DAC + Ipamorelin.

I’m also currently taking 1mg Reta weekly (I’m not large just to stay slim)

The CJC IPA is once in morning and once at night fasted.

Would this be a safe stack or unsafe. And why? Thank you.

Thought experiment, and I'm curious where this lands.

Say a lab gets funding to run one properly designed human trial on one research peptide. One compound, one year, real controls, real endpoints, published either way. They can only pick one.

Which one is the best use of that money?

I don't mean which one you find most exciting. I mean which one would change the most if somebody finally ran the trial. Which gap is costing us the most.

My pick: BPC-157

Not because I think it's the strongest compound. Because it's the biggest gap between how much it gets used and how little human data exists.

A 2025 systematic review screened 544 articles on it, and the human clinical trials are still basically absent. It just went through an FDA advisory panel where the agency's own scientists said there wasn't enough human safety data, and the panel recommended it anyway, 8 to 6.

So we've got a compound that's everywhere, with a huge animal literature, no real human trials, and a regulatory decision pending. One good trial would settle more arguments than anything else on the list.

Two other candidates I considered

LL-37. It has human randomized trials, all topical. Injectable use has no published human pharmacokinetics. One well-designed injectable study would close a gap that thousands of people are currently guessing at.

Epitalon. All the human outcome data belongs to Epithalamin, the extract. The synthetic has never had a human trial of its own. That's a clean, answerable question.

Your pick

Name one compound and say what the trial would settle. That second part is the interesting one.

And if you think the money is better spent proving one of these doesn't work, say that too. Negative results are worth as much and get funded a lot less.

For research use only. Not for human or veterinary consumption. This post is educational and is not medical advice.

Full doses and bloodwork are in the pinned cheat sheet.

Join the Discord.

Hallo, ich M42 habe in den letzten 11 Monaten 38kg abgenommen. Derzeit bei 83.5kg Körperfettanteil liegt bei etwa 12% die Haut am Bauch ist etwas schlaff. Ich habe vor einigen Monaten mit Peptieden begonnen und habe am 25.09. mit Testosteron E begonnen. Folgende Dosierungen gibt es:

Testosteron 90mg alle 2 Tage

Retatrutide 1,5mg alle 4 Tage

CJC-1295 mit Dac 1mg alle 4 Tage

Ipamorelin 0,9 mg jeden Tag in 3 Spritzen aufgeteilt

TB-500 1,5mg alle 2 Tage

BPC-157 1,5mg alle 2 Tage

GHK-Cu 2,5mg jeden Tag

Selank und Semax über Nasenspray bei Bedarf

Ich gehe mindestens 6 Tage ins Gym und Baller richtig gut Gewichte.

Soweit so gut jetzt kommen wir zu meinen Fragen:

1. HCG sinnvoll während der Testosteron gabe?

2. Wie lange sollte eine Kur dauern?( wollte 20 Wochen durchführen)

3. Sollten weitere Peptide vermieden werden? (Überlege HGH und Tesamorelin hinzuzufügen, eventuell auch MOTS-C)

4. Wie ist es beim beenden der Testosteron gabe, welche Medis sind sinnvoll welche wirklich nötig oder einfach weiter auf TRT fahren?

Vielen Dank für die Infos am liebsten offen und direkt😂😂