u/ANALyzeThis69420

So I got blue chew after having issues with staying hard. I had recently gone on Vyvanse and been increasing it for a year and a half. I’d say my issue started at 20 mg. It seemed to only get worse. I tried cock rings, taking more blue chew cialis I wish I had been able to figure out that you fake the shit at least two hours before… Seems like companies try to market us as sooner to get to buy it. Also blue chew stopped selling the crappy 6 mg I bought just recently. Why would they even sell that? I had been taking beet root and al these supplements to lower my blood pressure so initially I got vertigo. That’s partly why I stayed on that dose for a while before I started taking two. Eventually I got 20
Mg from amazon medical.

I think also i was in denial that I had a problem and of course went the non medical route initially. That’s what people do with ADHD and it is not good. The supplements help supplement the medication not just your diet.

I’ve been trying to incorporate more excercise and recently added nattokynase to go alongside my garlic pill. Finally I added Propionyl-L-Carnitine because it supposedly helps a lot of things for ED and just really boosts efficacy for cialis. I’m also looking at these combo pills like blue chew gold but I have a much cheaper one by “motivated.” It’s still not quite as potent it’s like $5 a pill. For the amount of sex I have it sounds totally worth it. For one sex with a limp dick becomes embarrassingly difficult and lame. You have to do all sorts of weird things just to stay half hard. The ingredients for connecting to your partner emotionally sound like they could just make the sex a lot better for both of you.

Oh I forgot to mention I joined a weight loss study that requires us to consume only 1500
Calories a day. I’m losing about two pounds a week but that has to hurt testosterone. I’m feeling like it isn’t tanking my metabolism so I’m staying with it. There are a lot more benefits in the study than just getting to practice this aspect.

Edit: I forgot to mention my Fitbit has been showing a heart rate of about 68 to 74 bpm and my heart rate variability is down to like 18 form a high of 22. Still super bad. This has to be from Vyvanse. It seems there is some link between the parasympathetic nervous system and ED caused by this. Any tips or insights?

I'm looking at things I can improve outside of what I was doing here for a while (reading articles and anecdotes here on diet.) Apparently the Oura Ring is endorsed by the Sleep Foundation for what it's worth and has much better data collection just by being a ring on the finger.

I just switched to a full face mask on my CPAP and it appears to have improved my sleep interruptions according to my fitbit. As we know poor sleep jacks up your insulin and eating.

It seems like you lose basically 40% muscle to 60% fat which is scary honestly. That’s based on my observation of minimal resistance training.

I’m wondering if lower carb dieting would offset this. I know body builders often rely on carbs to build muscle. Obviously a lot of us here are still eating a reduced protein diet somewhat.

Personally I would think eating lower carbs for a time while lifting heavy and maybe stairstepping your calories like Dave Fitness would help.

What do you think?

I’m not even sure if it’s claimed to help, but if it helps with heart rate variability it should help. I’m on Vyvanse and it both screws up HRV and worsens ED. The stimulators are still priced for “wellness moms”, but unlike going to yoga or cold plunges it’s just set it and forget it. Halp.

The Core Issue

Most people eat nowhere near the 25 to 30 grams of fiber recommended daily. And to meaningfully stimulate the gut hormones that suppress appetite, you'd typically need around 80 grams. That's not realistic for anyone.

The Finding

Researchers spent 15 years developing IPE (inulin-propionate ester), a modified fibre that delivers propionate, a gut-signaling fatty acid, directly to the large intestine. Just 10 grams a day appears to trigger the release of GLP-1 and PYY, the same hunger-suppressing hormones that Ozempic-style drugs artificially mimic. The European Commission officially authorized IPE for food use on June 9, 2026.

Why It Matters

This isn't a drug. It's a food ingredient that could show up in your smoothie or cereal bar within the next year. In one trial, none of the middle-aged participants taking IPE gained significant weight over six months, compared to 17% of the control group. A separate year-long trial in younger adults found an average increase of over a kilogram of fat-free body mass.

Limitations of Study

The weight-gain prevention trial in people over 40 was small. The fat-free mass finding in younger participants also couldn't confirm whether the gain was muscle specifically or other non-fat tissue. One outside researcher describes the overall evidence as mixed. The approval currently covers only the EU, and UK authorization is not yet in place.

Interesting Statistics

• 10g per day is the studied dose, a fraction of the 80g traditionally needed to stimulate appetite hormones through diet alone • 0% of the IPE group in one trial gained significant weight over 6 months vs. 17% of controls • Over 1 kg of fat-free mass gained on average in a year-long trial of younger overweight adults • EU approval allows up to 17g per 100g in cereal bars and up to 3g per 100ml in fruit smoothies • Imperial College holds exclusive EU market protection until June 30, 2031 • 12 years to secure European Food Safety Authority approval • Products are expected to reach EU shelves within 12 months

Useful Takeaways

IPE is framed as a preventive tool, not a weight-loss drug. Researchers are now exploring whether it could help people preserve muscle while on GLP-1 medications, or slow weight regain after stopping them. The only reported side effect is increased flatulence, which is standard territory for high-fibre intake.

TL;DR

A modified fibre called IPE triggers the same gut hormones as Ozempic at just 10 grams a day and is now approved for use in EU food products, with smoothies and cereal bars potentially on shelves within a year.

A review on how SGLT2 inhibitors target ectopic fat (specifically epicardial fat). Ectopic fat is the excess fat that gets stored in organ tissue. The paper outlines the different mechanisms of how it does so. For anyone not familiar with SGLT2 inhibitors, they cause you to urinate 60-80g of glucose, so it can cause a mild caloric deficit. It would be interesting to see a side by side comparison of how SGLT2i's compare to GLP1 reduction in ectopic fat.

I have already been a fan of l-theanine for a while now, but I started adding it to my coffee recently. Perhaps I should just move to straight decaf again. Sometimes I take l-tyrosine if my dosage is not working or I’m doing a demanding task.

One thing I thought about was serontonin levels being lower perhaps because of anxiety. I started taking 5-HTP to help with that. I personally thought I didn’t really have anxiety, but I guess I just power through it. You do need to excercise that muscle as part of it. However you can spiral and it seems you need a respor to grow sometimes. I did however hear an anecdote that serontin and dopamine can compete. Surely not.

Also I take methyl folate.