Hi - as someone who can't even visit a dentist and waited a year for a colonoscopy before they could do so with an anesthetist with Propofol, can I ask if cataract surgery is possible under Propofol? I asked a few providers and I get these sort of replies: it's painless, our numbing is really effective, it's quick, you won't move, 'let me reassure you", if you must Diazepam can be given. But WHY isn't Propofol administered by an anesthetist an option for those that are very anxious, don't want to experience it mentally? I can say that Diazepam has a very limited effect and Midazolam/Versed wasnmuch better but Propofol was brilliant, totally out cold. I did read somewhere that those with Restless Legs Syndrome needed Propofol to prevent moving around - so maybe I need claim just that to avoid being lecturered again?

I had my first eye done on July 22, about a month ago. The surgery went well, I went to the follow up exams the next day and a week later. My vision was blurry but continued to gradually get better. I was supposed to go for the second eye surgery two weeks later on August 5th but I still had inflammation and a bit of blurry vision so I had it moved up to next week, August 26. The surgeon's office had me come in today for another follow up. She said there is still inflammation and swelling and to continue the drops for another week. I'm at 20/25, I chose long distance monofocal lenses.

She also said she saw a little bit of scar tissue. Is this the same thing as PCO or is that different? When I had the operation I opted for the laser assisted surgery, they told me it makes recovery easier. I believe the laser assisted surgery breaks up the lens so it's easier to remove. She said the scar tissue can be removed with a laser about 3 months after recovery and it can be done in their office and takes maybe a minute if that long. Has anyone else here had this problem?

I had surgery last Friday and am wondering how long it took you to get your final result with the Puresee lens?

Scheduled next week for my right eye. Surgeon says Fuch's means I need to stick to monofocal lens. Also getting cornea layer transplant at the same time. 68 years old.

I have always been myopic (left (dominant) -7, right -5.5). Although I have unlined bifocal glasses I usually take them off to read or use my phone. Surgeon says "that means you probably won't be happy with distance vision", which agrees with the impression I get from reading a lot of the threads here.

So the plan is to have my right eye set to -2. Then in a few weeks set the left eye to -3. I think this gives me the best range for reading and using the computer. I realize a lot of people would go more -2.5 and -1.5, but that seems too far away for me.

Of course I will need glasses for distance. Not sure if I will want bifocals (with essentially plano in the bottom) or just for distance.

My other concern was losing my "superpower" of being able to read really small print by holding something 6" in front of my eye. Then I realized that I can get the superpower back by using strong readers (say 3.5). So hopefully I won't need readers for normal reading, just for super up close.

I have been reading this sub for awhile. Very impressed with the sharing, advice, and support here.

I want to order a backup pair of glasses so I don't have to go back to readers if I damage or lose my new progressives. The prices at Zenni are too hard to ignore $67 for some backups vs $300 for my new ones, but they won't let me enter a different NV Add for each eye, just one for both.

On my progressive lenses that I got for work required eye protection all day and so I don't have to keep grabbing readers has +0.25 Sph +0.50 Cyl and 68 Axis on the right eye, Plano Sph +0.25 Sph 51 Axis left eye. With that prescription I have 20/15 distance vision with both eyes and very low astigmatism (Toric IOL) which is pretty nice after 30 years of myopia with horrible astigmatism.

If I kept the +0.25 Sph on the right eye, Plano on the left with a +2.50 NV Add on both (has to be just one NV Add number) Will that small difference give me problems at close reading / viewing, or should I just bag the +0.25 Sph on the right eye and keep the NV Add at +2.50, losing a bit of the tasty crispness in my right eye for the inexpensive backups?

I had RK in the 1980s. I have 16 cuts on each eye. Today I learned that I “potentially” “do” have glaucoma. I also learned that an LAL lens is the best, really only, solution for my vision. I’m processing this over a cortado. I’ll get second opinion next week. Then I’ll go from there. Not panicking.

For blf iol's like pursee what % of blue light do they block? Cheers

Fellow myopics with astigmatism who underwent cataract surgery, which toric monofocal correction—near, far, or intermediate—did you choose? Happy or regretful?

My dad has ckd , diabetes and suffering from diabetic retinopathy I need genuine response on the treatment provided from this hospital also if there are any good eye specialist do let me know . Ps. Don't suggest any long queue hospitals.

Hey everyone,

I am 18 years old and was born with a congenital central cataract in both eyes, along with a severe astigmatism of -5.25 dpt. Since this condition has been present since early childhood, my visual system developed with it, which naturally adds some complexity to evaluating potential surgery.

My daily vision is quite challenging. In dim or indoor lighting, my vision tests at around 50 to 60 percent with my glasses. However, as soon as I go outside into bright daylight, the light hits the central cloudiness and creates a severe, blinding glare. Driving in full sunlight is extremely difficult because the brightness washes out the entire image - making my practical outdoor vision much lower than what the indoor tests show.

So far, my doctors have been cautious about recommending IOL surgery. Their hesitation is very understandable: since I grew up with this central opacity, there is a medical concern about amblyopia - meaning it is unclear how much maximum sharpness my brain can process, even with a clear artificial lens. Additionally, surgery at age 19 means losing natural accommodation. Another important factor is that my glare sensitivity has only been discussed subjectively so far; no quantitative straylight or glare test has been performed yet to measure the actual optical impact of bright light on my vision.

However, a very interesting effect happens every time my pupils are dilated with drops at the clinic. Once the pupil is wide, my distance vision transforms completely. I suddenly see crisp details, vivid color contrasts, and sharp textures in the distance that I normally cannot perceive. Since my cataract is strictly central, the wider pupil allows light to pass around the cloudy center through the clear outer edge of my natural lens.

I haven't had the chance to discuss this specific "dilation effect" with my doctor yet, as my previous appointments were quite brief. My next step is to get a second opinion at another university eye clinic, where I plan to request a quantitative glare test and discuss what this dilation effect implies for my retina's potential.

I am leaning towards surgery - ideally with toric IOLs to also address the -5.25 dpt astigmatism - because I would gladly trade natural accommodation for clear, glare-free distance vision. Has anyone here with a lifelong congenital cataract experienced a similar dilation effect? How did your doctors view the situation, and did IOL surgery help you achieve stable vision in bright light? I would really appreciate your thoughts and experiences.

Hi Everyone,

I am scheduled for Cataract surgery on right eye (my left eye is 202/20 for distance and +0.25 for reading) on Thursday, Aug 27th and I am getting the Light-Adjustable lenses (LAL) which was recommended because of my prior LASIK.

I need to go into the office on Tuesday, Sept 1st (5 days post-op) for for work for a customer presentation. I know LAL comes with strict requirments (need to wear Uv-protctive glasses, but would this be feasible for me to go into the office short 30 minutes presentation. I don't plan to drive and will be taking public transportation.

For those of you who chose LAL and work in office settings:

• How was working on Monitor/screns so soon after surgery while wearing UV glasses?
• Since on eye is being done intially, did the visual diiference between eye makes working or commuting difficult in those first few days?

I plan on to clear everyting with my surgeon at my post-op visit (Aug 28th) but wanted to hear first hand experiences from anyone who has had LAL surgery and had to go into office so soon after surgery.

Thanks!

I have mild astigmatism (-0.5 to -0.75) and presbyopia. I want the clearest possible mid and distance vision and least chance of complications. I don’t mind reading glasses but of course the trifocal lens is attractive if I could stop wearing glasses completely.
I cannot do mono vision.

I have eyelash issues, I have alopecia and it affects my right eyelashes, so they are always falling out in my eye, curling in my eye, I have always rubbed my right eye so much cause it was so bad. So I had my right eye done, left eye was 6 months ago, and it's been a pain. I have to go every few days to have the Dr. pluck the ones in the corner of my eye, he says they are also in the tear duct area, that little mound of flesh. Anyway, my eyelashes are always catching on the guard, then I have to remove it, tape it back, I will likely wear it to sleep for a couple more months, cause I know I will rub it when I'm sleeping, I try to get in the tape when I'm sleeping. I can't find one that is deep enough to not touch my eyelashes, I've tried a few of the clear plastic ones on Amazon, they don't work. Anyone out there have this issue, have long lashes, can suggest something that I can tape on and will work? Thanks.

Let me start by saying I have alternating esotropia. I was nearsighted in one eye and farsighted in the other, and I can control which eye I use.

In the nearsighted eye, I had a cataract. I had a lens replacement. Since then, my vision did improve. However, when I use that eye to focus on close objects such as scrolling my phone or reading a book, it's as if the eye gets stuck in close focus. Looking up from the phone, for example, everything else now has a "shadow" or slight doubling. It won't clear up until I sleep for the night.

Anyone else have this? What can I do?

Is this normal? I had cataract surgery three days ago, and everything I read suggests that the vast majority of people are able to see well and function almost immediately after surgery. I’m not expecting 20/20 vision, or even to not need glasses, but I’m not even at 20/40 vision in that eye. And, there’s just enough sight in my operated eye that, uncovered, it distracts from my better eye when I drive. Previously my brain just ignored that eye. Has this happened to anyone else? Have you had surgery and then just been very blurry for days after?

I have/had no other eye issues in my operated eye except a very bad cataract. I’ve only been to one follow up where the doctor was ok was the healing, but I’m terrified. Have you ever experienced a surgery where it took longer than normal to heal? And, for all the people who will say everyone heals differently, I do know that. Im hoping to hear from at least one person who can tell me that they’ve been through something similar. I’m real scared and afraid it won’t improve.

I will eventually need cataract surgery on my good eye, and I was going to get it in a couple of weeks, but now I’m tempted to cancel. I’m worried that I’ll become effectively blind.

Tell me about it if you’ve had this experience.

There have been discussions on here about eyedrops (Lanosterol) that might reverse cataracts in the future instead of the need for surgery. Work has been done with rats and dogs. Thought I’d share this YT video today about the start of a US clinical trial with Lanosterol, ZOC2017217.

I found ophthalmologist Dr. Marc Shomer’s video informative about the history of results from trials in the past as well as the use and warnings of castor oil. He also lists legit recommendations for protecting your eye sight now.

Lanosterol US clinical trial

Have a family member who has stage 3 cataracts in one eye and stage 4 cataracts in the other. The cataracts have progressed pretty fast unfortunately. We have an appointment with a Montefiore surgeon, but while I’m sure the doctor that was recommended is great, I am concerned about surgeon experience in complex cataracts.

Does anyone have suggestions on surgeons, within/close to NYC area? Also we’re trying to get this done ASAP because we’re concerned about complications with time, so would appreciate if you could provide any insight on wait times. Also we are generally open to traveling if we find a good surgeon.

I can’t find anything about people having this issue, so asking here.

I had surgery getting LAL+ lenses in both eyes. Everything went fine. We did one adjustment in each eye, then at the 3rd adjustment appointment I decided that I liked my vision as we had it, so we did nothing. The follow 2 weeks we did the 2 lock sessions. I was completely happy with my vision. Then over the next couple of months I slowly became more nearsighted in both eyes!

I specifically set my vision so that I could drive and watch TV without glasses, but would still wear reading glasses. I had been wearing reading glasses for years so that was fine. It was really great to finally having good distance vision after being nearsighted my entire life.

Well now I’ve lost that. The TV is blurry and I need glasses to drive again. And I’m hesitating to get correct glasses fearing that my vision will continue to change.

Anyone have similar experience or information? Thanks

Persistent blurred vision, poor contrast and ghosting at all distances after RayOne Galaxy Toric IOL — little improvement with refraction

TL;DR

I am a 34-year-old male. I had left-eye cataract surgery on April 13, 2026, with implantation of a RayOne Galaxy Toric IOL. At the time of writing on August 16, 2026, I am only about four months post-op. Since the first postoperative day, the eye has never produced a genuinely sharp or comfortable image.

• Distance vision is continuously blurred, low-contrast and smeared. Refraction improves it only slightly.
• Intermediate vision is poor. I can sometimes barely recognize or read text, often partly by guessing, but I would not call it usable.
• Without optical correction, normal-sized phone text is completely unreadable and not recognizable as individual letters; it appears blurred out.
• During my examination on August 15, 2026, the doctor tested my near vision using that day's correction (plano/−1.00 × 170) plus approximately +1.75 to +2.00 D ADD. Near text was then clear and comfortable.
• Letters have ghost images or light streaks, and vision feels like looking through a thin transparent film.
• Blinking and artificial tears do not meaningfully change the image.
• My right eye has its natural lens and sees 20/20 without correction.

Depending on the hospital, the left eye has been recorded between 0.4 and 0.7 with correction. The 0.7 result involved recognizing blurred letters partly by guessing; functionally, the image feels closer to approximately 0.5 and is much less comfortable than 0.7 suggests.

After visiting three hospitals, I still do not have a root-cause explanation. The possibilities I am considering include IOL tilt/decentration or another internal optical problem; clinically significant vitreous scatter, although I currently think this is less likely; or an inaccurate IOL power/toric correction, although this is unconfirmed because residual refractive astigmatism has generally measured only about 0.5–1.0 D depending on the examination. I currently consider the cornea less likely because the latest map was described by the doctor as fairly regular and symmetric without an obvious focal hot spot. I understand that map colors depend on the display scale and do not completely exclude a corneal contribution.

My main questions are:

1. Has anyone experienced a similar problem after an EDOF toric IOL? What was eventually found, and how was it managed?
2. Based on the symptoms and measurements below, what hypotheses would you consider in my case?
3. If you have seen a similar case, which specialist or examination actually helped identify the root cause?

Surgery on April 13, 2026, IOL and preoperative baseline — Hospital A

Hospital A performed my surgery on April 13, 2026, and holds the preoperative records. At the time of writing on August 16, 2026, I am only about four months post-op.

The implanted IOL is:

• RayOne Galaxy Toric, model RAO615X
• Spherical equivalent: +17.00 D
• Sphere: +15.87 D
• Cylinder at the IOL plane: +2.25 D
• Manufacturer-listed near add: +3.00 D
• Manufacturer-listed intermediate add: +1.50 D
• Optic diameter: 6.0 mm
• Target refraction: plano

The following measurements were taken with a Heidelberg Engineering ANTERION on March 14, 2026. These were obtained approximately one month before surgery; March 14 was not the surgery date:

• Axial length: 25.06 mm
• Total corneal astigmatism: 2.53 D at 94°
• Corneal HOA RMS at 5 mm: 0.22 µm, almost identical to the right eye at 0.21 µm
• Spherical aberration: 0.07 µm
• Pupil diameter: 5.2 mm
• Central corneal thickness: 513 µm

The preoperative cornea therefore appeared relatively regular. Multiple IOL formulas converged around +17.00 D for a plano target, so a major spherical IOL power error seems unlikely.

Latest examinations

Hospital C — August 7, 2026

A ZEISS VISUREF autorefractor/keratometer measured −0.25/−0.50 × 160, with approximately 2.25 D of corneal astigmatism. Subjective testing showed 20/40 both uncorrected and corrected; the record from this hospital listed a +1.75 D near ADD. Posterior OCT/RNFL and fundus examination found no explanatory macular or optic-disc lesion. B-scan ultrasound showed vitreous opacity without retinal/choroidal detachment.

The doctor said that nothing further could be done or offered. However, this visit did not include corneal tomography, whole-eye/internal aberrometry, quantitative IOL tilt/decentration analysis, retroillumination, contrast-sensitivity or straylight testing. My personal impression, which may be wrong, is that the hospital preferred not to pursue riskier intervention; I do not think the performed tests established that no treatable cause exists.

Hospital B — August 14, 2026

An OCULUS Pentacam showed regular corneal astigmatism of 2.4 D at 84.4° and a thinnest point of approximately 515 µm. Clinical testing showed 0.2 uncorrected and 0.4 corrected with −1.50/−0.50 × 160.

The doctor said PRK would probably improve vision only to the current corrected level, approximately 0.4. The hospital focused on corneal laser and did not discuss detailed evaluation of the internal IOL–capsule system. The examination was not dilated. Communication was through an interpreter, who suggested amblyopia; this was not presented to me as a diagnosis confirmed and explained by the doctors, and no functional testing was performed to establish it.

Hospital A — August 15, 2026

Postoperative Heidelberg Engineering ANTERION measurements showed 2.66 D of total corneal astigmatism at 88°, corneal HOA RMS 0.28 µm at 5 mm, pupil diameter 7.2 mm and pupil-center offset 0.31 mm at 278°. The surgeon considered the corneal map regular.

The report incorrectly classified the eye as “No surgery, Phakic, Vitreous only,” and no dedicated IOL Analysis page was obtained. I therefore still have no validated quantitative measurement of IOL tilt or decentration.

Clinical testing recorded 0.7 both uncorrected and corrected with plano/−1.00 × 170, but I was recognizing blurred letters partly by guessing. The surgeon reported no PCO, no obvious IOL tilt on routine examination and approximately 1.00 D residual astigmatism. PRK was not recommended because the expected gain was small and it could make vision worse. IOL exchange was also not recommended because of surgical risk. I am not requesting exchange without evidence, but I would like assessment by a surgeon experienced in complex IOL exchange before that option is dismissed.

During the same examination on August 15, 2026, the doctor tested my near vision using that day's correction (plano/−1.00 × 170) plus approximately +1.75 to +2.00 D ADD. Near text was then clear and comfortable.

Home pinhole experiment — non-standard

I repeated this informal test using several homemade apertures and rotated them in different directions.

• At approximately 1.5 meters, a 2–3 mm aperture could make small computer text comfortably readable; under the best conditions, subjective sharpness reached approximately 85–90% of the right eye. Larger openings brought back blur and light streaking.
• At phone-reading distance, normal text was unreadable without optical correction. A 1–2 mm aperture allowed some reading, although still with difficulty; at approximately 3 mm it became distinctly blurred again.
• Through a small aperture, the central area remained readable, nearly circular and symmetrical but slightly darker/softer. Part of the periphery appeared brighter/sharper, creating a non-standard C-shaped appearance.

This was not standardized and does not prove IOL decentration or localize the cause.

What seems less likely, but is not absolutely excluded

• Major IOL power error: preoperative calculations and current refraction are relatively close to plano.
• Ordinary refractive error alone: multiple spectacle corrections provide little improvement and do not remove the ghosting.
• Major macular structural disease: OCT and fundus examination did not identify an explanatory lesion.
• Gross corneal irregularity as the sole cause: preoperative and latest ANTERION corneal HOA values were relatively low. The doctor described the latest map as fairly regular and symmetric, mainly blue with some symmetric yellow areas and no obvious focal orange/red hot spot. Map colors depend on the display scale, so a corneal contribution is not completely excluded.
• Dry eye as the main cause: blinking and artificial tears do not change the characteristic image.
• PCO or another obvious abnormality of the capsule holding the IOL: the doctor examined this carefully on August 15, 2026, and found none. I therefore no longer consider it a leading possibility. A very subtle abnormality cannot be excluded completely because I do not have photographs for comparison.
• Severe amblyopia: during the August 15, 2026 examination, near vision became clear with plano/−1.00 × 170 plus approximately +1.75 to +2.00 D ADD. Vision can also improve substantially through a small aperture. Mild amblyopia or another functional abnormality has not been excluded; a normal OCT alone cannot diagnose or exclude amblyopia.

What remains uncertain

• Whether the IOL is tilted. This has not been measured. I do not know whether the ANTERION can perform the required analysis, and my doctor did not appear certain about this.
• The range-of-vision optical design of the RayOne Galaxy may not suit my eye. If this were confirmed, one possible option might be exchange for a different IOL—but only after every other reasonable cause has been excluded.
• The IOL power or toric correction may be inaccurate, although this remains unconfirmed because residual refractive astigmatism has measured only approximately 0.5–1.0 D, depending on the examination.
• Could vitreous scatter be contributing? I currently consider it less likely because it has not been quantified, and none of the doctors seemed to suspect it.
• Residual astigmatism or another corneal optical problem currently seems less prominent because the latest corneal map was considered fairly regular and spectacle correction—including correction of the astigmatism—improved vision very little.
• A subtle retinal or optic-nerve abnormality not visible on structural OCT.

My goal and request for advice

I believe my left eye may have better visual potential than my current day-to-day vision suggests. I am not demanding guaranteed 20/20 vision. I want a stable, readable and comfortable image, ideally without glasses or with as little dependence on them as possible.

PRK is not currently an option I am considering because the doctors expect little benefit or possible worsening. I also do not want any irreversible procedure merely as a trial. I am uncomfortable treating “amblyopia,” “vitreous opacity,” “the IOL looks fine” or “nothing more can be done” as final explanations without confirming tests.

Has anyone managed or experienced a similar case? What was the final cause, and how was it managed? Based on all of these data, which hypothesis would you consider most likely? Which specialist or examination helped confirm the cause in your experience?